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Diagnosis and Management of Endocrine-related Tumors by Robert B. Page, Richard J. Santen (auth.), Richard J. Santen

By Robert B. Page, Richard J. Santen (auth.), Richard J. Santen M.D., Andrea Manni M.D. (eds.)

Patients with a number of tumors current to the health care professional due to medical manifestations of hormones secreted in extra. This phenomenon attracted the investigative curiosity of such pioneers as Harvey Cushing who well-known that pituitary tumors can cause acromegaly and Charles Mayo who linked hyper­ stress with adrenal medullary neoplasms. present int~rest in endocrine-related tumors has intensified as a result explosive improvement of more moderen methodol­ ogy for his or her research. particular measurements of secretory items, hybridization assays to spot items of genomic translation and quantitative evaluation of tissue hormone receptors have supplied technique of characterizing and accurately following sufferers with endocrine-related tumors. remedies established upon those advances are speedily proliferating. the present quantity makes an attempt to synthesize a lot of this fresh details with the target of delivering a legitimate foundation for making medical decisions relating to analysis and administration. Tumors of endocrine glandular tissues generally confront working towards physi­ cians with tricky administration difficulties. a number of distinct gains of those tumors necessitate collaboration between quite a few forte disciplines with a view to unravel those difficulties and to supply a excessive point of medical care. for instance, endocrine neoplasms secrete energetic hormones or hormone precursors which produce medical manifestations so much popular to endocrinologists. definite thera­ pies equivalent to radioactive iodine for thyroid melanoma make the most of the hormone­ responsiveness of those tumors to facilitate therapy. those points require participants expert in endocrinology to enforce complicated diagnostic and thera­ peutic maneuvers.

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6a). Note small homogeneous pituitary gland, flat diaphragm sella, water density over superior border of gland and straight vertical pituitary stalk (white arrowhead) . Figure 14b. Microadenoma of pituitary gland. Note hypodense area within pituitary at arrowhead. Figure 14c. Macroadenoma with suprasellar extension. 21 placed in two groups (enclosed and invasive). Hardy [120] defined enclosed tumors as tumors which remain 'within the anatomical confines of the osteoaponeural sheath of the sella turcica'.

In: Neurosecretion and Brain Peptides, Martin JB, Reichlin S, Bick KL (eds). New York: Raven Press, 1981, pp 63-74. 30. Bugnon C, Fellmann D, Gouget A, Cardot J: Corticoliberin in rat brain: immunocytochemical identification and localization of a novel neuroglandular system. Neurosci Letters 30: 25-30, 1982. 31. Palkovits M: Neuropeptides in the median eminence: their sources and destinations. Peptides 3: 299-303, 1982. 32. Page RB: Pituitary blood flow. Am J Physiol243: E427-E442, 1982. 33. Bjorklund A, Moore RY, Nobin A, Stenevi U: The organization of tubero-hypophyseal and reticulo-infundibular catecholamine neuron systems in the rat brain.

Usually the tumor has herniated sufficiently high into the hypothalamus to be visualized through the foramen as monro which is enlarged and there is no need to transect either the fornix which may lead to memory deficits, or the strio-thalamic vein which may lead to infarction of ipsilateral caudate nucleus. If exposure is limited, it may be carried posterior to the strio-thalamic vein. In this region the tinea choroidea may be incised without damage to the internal cerebral vein and the third ventricle may be entered posterior to the foramen of Monro [176].

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