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Dementia: Presentations, Differential Diagnosis, and by V. Olga B. Emery PhD, Thomas E. Oxman MD

By V. Olga B. Emery PhD, Thomas E. Oxman MD

During this new version of the acclaimed Dementia: displays, Differential analysis, and Nosology, V. Olga B. Emery, Ph.D., and Thomas E. Oxman, M.D., collect a exceptional workforce of clinical authorities—including many that have performed seminal examine during this field—to talk about the spectrum of dementing issues and clarify their overlap, displays, and differential analysis. The chapters current unique information in addition to fabric from the authors' scientific reviews. present class structures are evaluated and converted to higher account for universal displays of dementia. completely revised, up-to-date, and increased, the second one version contains new fabric on neuroimaging, genetics, the position of irritation in Alzheimer sickness, retrophylogenesis in Alzheimer reminiscence, and on AIDS dementia. moreover, each one bankruptcy encompasses a new part entitled describing medical purposes.

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Extra resources for Dementia: Presentations, Differential Diagnosis, and Nosology (The Johns Hopkins Series in Psychiatry and Neuroscience)

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Brains from persons with DAT demonstrate greater neuronal loss in frontal and temporal regions (Mountjoy et al. 1983). Subcortical nuclei including the locus ceruleus and basal nucleus of Meynert also show greater cell loss in DAT, but this may be more typical in those with a younger age of onset (Iversen 1987). The dominant theory of DAT has been related to loss of cholinergic cells, but recent ~ndings of upregulation of choline acetyltransferase activity in MCI and mild DAT may extend this model to include compensatory changes (DeKosky et al.

J. Li, and J. Jones. 2000. A functional MRI index as a biological marker for Alzheimer’s disease. Paper read at American Academy of Neurology. F. Friston. 2000. Voxel-based morphometry: The methods. NeuroImage 11:805–21. D. 1986. Working Memory. Oxford: Clarendon Press. J. Hitch. 1994. Developments in the concept of working memory. Neuropsychology 8:485–93. B. 1993. The aging mind: Potential and limits. Gerontologist 33:580–94. M. Baltes. 1990. Psychological perspectives on successful aging: The model of selective optimization with compensation.

1999, 2001). This contrasts with the 1–2% per year rate at which DAT develops in the normal elderly population. Petersen and colleagues (1999) reported that up to 40% of individuals diagnosed with MCI convert to DAT within four years, and Morris et al. 5 years. Given the high likelihood of progression to dementia of the Alzheimer type in individuals with mild cognitive impairment, early identi~cation and diagnosis may be important (Almkvist and Winblad 1999). While it is not yet clear who will or will not convert to DAT, several potential risk factors have been identi~ed.

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