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Calcium Metabolism, Bone and Metabolic Bone Diseases by C. A. Baud, J. A. Pouëzat (auth.), Friedrich Kuhlencordt Dr.

By C. A. Baud, J. A. Pouëzat (auth.), Friedrich Kuhlencordt Dr. med., Hans-Peter Kruse Dr. med. (eds.)

The X. ecu Symposium on Calcified Tissues came about in Hamburg from sixteenth to 21 st September 1973. The monetary backing got here from the Gesund­ heitsbehorde der Freien und Hansestadt Hamburg (President Dr. Zylmann), the Bundesministerium fur Jugend, Familie und Gesundheit, and from undefined. This made it attainable to hold out the medical application, to ask numerous ecu and non-European scientists, and to arrange a social software designed to set up own touch among delegates and to make viewers familiar with our nation. within the identify of the organizing committee, we herewith exhibit our warmest appreciation of all of the aid given to us. Professor Bartelheimer, because the consultant of the scientific school and on behalf of the President of the collage of Hamburg, welcomed the Parti­ cipants within the symposium to our urban. The heritage of those conferences started in Oxford in 1963, with the 1st ecu Bone and enamel Symposium, prepared via H. J. J. Blackwood, B. E. C. Nordin, and Dame Janet Vaughan. the belief was once to present in Europe an establishment simi­ lar to the yankee Gordon examine meetings of Bone and teeth. After the U. ok. , the host international locations have been Belgium, Switzerland, the Netherlands, France, Sweden, Italy, Israel and Austria. the second one symposium in Liege already bore the current name.

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J. cell Biol. 20, 165-173 (1964). CRABB, MORTIMER: Two-dimensional microdensitometry. Brit. dent. J. 122, 337-343 (1967). W. : Preparation of undecalcified bone sections for auto- and microradiography. Ca. Tissue Research ~, 23-30 (1972). , KOFSKY: Simplified two-dimensional microdensitometry. Nature 202, 1196-1200 (1964). 39 On Calcium Metabolism during Immobilization H. BOHR It is well known that decisive changes in metabolism take place during immobilization. Thus it was shown by DEITRICK, WHEDON and SHORR (1948) that excretion of calcium in the urine and feces increased, resulting in a negative calcium balance.

67 mg/day). Less is known about what happens in kidney. , 1961), but no data are available to evaluate this quantitatively, as in Eqs. 3-6. Fig. 3 is a regulatory diagram showing the addition of parathyroid function. In the model, a negative error is drawn as actuating the parathyroid glands to release hormone that acts on bone and kidney. In addition, as in Fig. 2, both positive and negative errors are shown acting directly on bone and kidney. Bone responds to negative errors by increasing vo- more than Vo+ and to positive errors by decreasing v o - more than vo+.

They commented on the fact that the animals with parathyroid autografts, in the quantitative terms of the analysis employed, were not quite "normal". In particular, regulation by bone seemed less efficient, though the overall capacity seemed unchanged, viz. =O, vowas about the same in the normal rat and that bearing a pa~athyroid autograft. However, the latter tended to handle a somewhat smaller proportion of the incoming load (Si) via diminution of bone calcium resorption. 9 mg/day when Si was O.

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