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Approaches to Bronchitis by Marianna D. Gaça

By Marianna D. Gaça

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The long duration of exposure to O3, PM10 and nitrogen dioxide (NO2) has been associated with chronic respiratory disease and reduced lung function, and with increased incidence of colds (Gilmour et al. 2006; Trasande and Thurston, 2005 ; Brunekreef and Holgate, 2002) In São Paulo, Brazil, Saldiva et al. (2008) found that vascular and pulmonary diseases are associated with long-term exposure to air pollution. Putting the hypothesis that inflammation of the lung epithelium and endothelium, resulting in airway and vascular pathology, are due to exposure to polluted air.

1992. Production and characterization of monoclonal antibodies to three infectious bronchitis virus serotypes. Avian Dis. 36, 903-915. , 1998. Serotype identification of avian infectious bronchitis virus by RT-PCR of the peplomer (S-1) gene. Avian Dis. 42 (2), 275-284. , 1991. Infectious bronchitis. , Jr. ), Disease of Poultry, 9th ed.. Iowa State University Press, Ames, Iwoa, pp. 471-484. , 2000. Identification of avian infectious bronchitis virus by direct automated cycle sequencing of the S-1 gene.

Purpurea roots contained relatively potent activity against influenza virus and HSV. In contrast, the antiviral activities of E. angustifolia roots were found in the ethanol and ethyl acetate fractions and included antirhinovirus activity, which was not detected in the aqueous fractions. E. pallida root extracts showed no antiviral activity whatsoever in any of the solvent fractions, in spite of the presence of the usual chemical markers for Echinacea species. Thus, in addition to the variation in activity among different species and extracts, there was no correlation between antiviral activity and relative content of caffeic acid derivatives, polysaccharides and alkylamides, suggesting that these compounds are not individually the active ingredients.

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