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Alcohol And the Gastrointestinal Tract: Special Issue: by M. V. Singer, D. Brenner

By M. V. Singer, D. Brenner

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Additional info for Alcohol And the Gastrointestinal Tract: Special Issue: Digestive Diseases 2005

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Via normal distribution and evaporation, salivary acetaldehyde may reach all target tissues of the upper aerodigestive tract. Consequently, it is suggested that the major part of the carcinogenic role of alcohol is caused by its first metabolite, acetaldehyde, which is microbially produced [48]. Dig Dis 2005;23:195–203 199 Alcohol Effect on DNA Repair Mechanisms Several authors have suggested that alcohol may have an effect on DNA repair mechanisms [8, 11, 86, 87].

Microbial salivary acetaldehyde production shows high inter-individual variation, but there exists a significant positive correlation between salivary ethanol and acetaldehyde levels. Moreover, in vivo salivary acetaldehyde levels correlate very significantly with the levels that are produced in vitro. This offers the opportunity to use the in vitro salivary test as a tool to investigate possible variables which might influence salivary acetaldehyde production. Salivary acetaldehyde levels after ethanol intake strikingly exceed those known to be derived from endogenous metabolism of ethanol [85].

J Nutr 1999; 129: 1945–1950. : Effects of chronic dietary beer and ethanol consumption on experimental colonic carcinogenesis by azoxymethane in rats. Cancer Res 1987; 47: 1551–1559. 57 Pitchumoni CS: Pathogenesis of alcohol-induced chronic pancreatitis: facts, perceptions, and misperceptions. Surg Clin North Am 2001;81:379–390. Animal Models and Their Results in Gastrointestinal Alcohol Research 58 Haber P, Wilson J, Apte M, Korsten M, Pirola R: Individual susceptibility to alcoholic pancreatitis: still an enigma.

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